this post was submitted on 17 Jun 2026
1 points (55.6% liked)
Friendly Carnivore
102 readers
13 users here now
Carnivore
The ultimate, zero carb, elimination diet
Meat Heals.
We are focused on health and lifestyle while trying to eat zero carb bioavailable foods.
Keep being AWESOME
We welcome engaged, polite, and logical debates and questions of any type
Purpose
- lifestyle
- food
- Science
- problems
- Recipes
- Sustainability
- Regenerative lifestyle
Rules
- Be nice
- Stay on topic
- Don't farm rage
- Be respectful of other diets, choices, lifestyles!!!!
- No Blanket down voting - If you only come to this community to downvote its the wrong community for you
- No LLM generated posts . Don't represent machine output as your own, and don't use machines to burn human response time.
Other terms: LCHF Carnivore, Keto Carnivore, Ketogenic Carnivore, Low Carb Carnivore, Zero Carb Carnivore, Animal Based Diet, Animal Sourced Foods
Meta
If you need to block this community and the UI won't let you, go to settings -> blocks you can add it.
[Meta] Moderation Policy for Niche Communities
founded 11 months ago
MODERATORS
you are viewing a single comment's thread
view the rest of the comments
view the rest of the comments
The argument is that plant sterols interfere with cholesterol function, impacting systems all over the body including vitamin d production.
https://doi.org/10.1159/000337881 -
Effect of Plant Sterols on Vitamin D Absorption
Cholecalciferol (vitamin D 3 ) is either obtained from the diet or produced photochemically in the skin from 7-dehydrocholesterol, which is produced in relatively large quantities from cholesterol in the gut1 and is accumulated in the skin [1]. Since plant sterols and stanols alter micelle formation and decrease absorption of cholesterol, it is possible that absorption of fat-soluble nutrients including fat soluble vitamins is also affected.The objective was to examine if plant sterols interfere with the absorption and possibly the synthesis of vitamin D3 . A randomized study was conducted in 40 apparently healthy adult volunteers aged 18–60 years who received orally either 25000 IU vitamin D 3 (group A, n = 20) or 25000 IU vitamin D3 together with 2 g plant sterols (Group B, n = 20). Levels of Vitamin D 3 , 25-hydroxyvitamin D 3 , calcium, phosphorus, magnesium, cholesterol and parathyroid hormone were measured in blood taken immediately before consumption of Vitamin D 3 and at 12 h, 24 h and 168 h (7 days) after administration of Vitamin D 3 . Serum vitamin D 3 concentration increased significantly in both groups at 12 h and 24 h and 25-hydroxyvitamin D3 at 168 h after the consumption of vitamin D. Group B had lower vitamin D 3 levels 12 hours and 24 hours after administration and lower 25-hydroxyvitamin D 3 levels at 168 h after administration.
Hence, inhibiting the absorption of cholesterol may also affect the absorption of vitamin D 3 . Long term (> 4 weeks) administration of sterols is needed to investigate the impact on Vitamin D absorption and consequently its metabolism.
It's well known seed oils will lower LDL cholesterol, this is a direct demonstration of their impact on the bodies hormone system, and cholesterol functioning. This is not a good thing.
Cancer happens all the time, right now at this very second both you and I have cancer cells in our bodies - typically not enough to overwhelm us and we survive (typically). The magical power is keeping the body healthy so immune function is excellent, so that the homeostatic machinery can do its job.
https://doi.org/10.1111/joim.12496
Avoidance of sun exposure as a risk factor for major causes of death: a competing risk analysis of the Melanoma in Southern Sweden cohort
Results Women with active sun exposure habits were mainly at a lower risk of cardiovascular disease (CVD) and noncancer/non-CVD death as compared to those who avoided sun exposure. As a result of their increased survival, the relative contribution of cancer death increased in these women. Nonsmokers who avoided sun exposure had a life expectancy similar to smokers in the highest sun exposure group, indicating that avoidance of sun exposure is a risk factor for death of a similar magnitude as smoking. Compared to the highest sun exposure group, life expectancy of avoiders of sun exposure was reduced by 0.6–2.1 years.
Conclusion The longer life expectancy amongst women with active sun exposure habits was related to a decrease in CVD and noncancer/non-CVD mortality, causing the relative contribution of death due to cancer to increase.
More sun exposure more cancer, but longer life....... so doing everything with a focus on avoiding all types of cancer may (may) be counter productive. You will note Dr Mason didn't tell you to not use sunscreen, and didn't tell you to stop avoiding the sun, he only talked about vitamin D function and how carnivores report not getting sunburn - it's not a recommendation for behavior.
The study you linked is about the effect of phytosterols on Vitamin D adsorption from an oral source.
It says nothing about Vitamin D production from sunlight.
And the study involved a single dose of Vitamin D given, to 40 people.
So the study itself says "for actual evidence, we'd need more data".
The point was to demonstrate plant sterols have a impact on the bodies cholesteral systems including vitamin d production and absorption.
Literally every paper ever written says that. We always need more studies, that's the nature of curiosity. In fact professors have to admonish grad students not to add that to papers since its just filler at this point.
I'm not sure what your core thesis is: Vitamin D is not relevant in sun exposure?
https://doi.org/10.1016/j.jid.2017.04.040 -
Oral Vitamin D Rapidly Attenuates Inflammation from Sunburn: An Interventional Study
The diverse immunomodulatory effects of vitamin D are increasingly being recognized. However, the ability of oral vitamin D to modulate acute inflammation in vivo has not been established in humans. In a double-blinded, placebo-controlled interventional trial, 20 healthy adults were randomized to receive either placebo or a high dose of vitamin D3 (cholecalciferol) one hour after experimental sunburn induced by an erythemogenic dose of UVR. Compared with placebo, participants receiving vitamin D3 (200,000 international units) demonstrated reduced expression of proinflammatory mediators tumor necrosis factor-α (P = 0.04) and inducible nitric oxide synthase (P = 0.02) in skin biopsy specimens 48 hours after experimental sunburn. A blinded, unsupervised hierarchical clustering of participants based on global gene expression profiles revealed that participants with significantly higher serum vitamin D3 levels after treatment (P = 0.007) demonstrated increased skin expression of the anti-inflammatory mediator arginase-1 (P = 0.005), and a sustained reduction in skin redness (P = 0.02), correlating with significant expression of genes related to skin barrier repair. In contrast, participants with lower serum vitamin D3 levels had significant expression of proinflammatory genes. Together the data may have broad implications for the immunotherapeutic properties of vitamin D in skin homeostasis, and implicate arginase-1 upregulation as a previously unreported mechanism by which vitamin D exerts anti-inflammatory effects in humans.
Yes, its oral, but it demonstrates that vitamin D is a necessary part of the bodies sun exposure mechanisms. i.e. its produced on demand to treat the damage the sun is doing to the skin.